Episode 65 – Cholesterol: An Overview

Speaker 1: 00:00 This is Dr Chad Edwards and you’re listening to podcast number 65 of against the grain.

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Speaker 1: 01:01 This is the Super Tall Marshall Morris, and today I am joined with the most incredibly humble Dr Chad Edwards and Dr Edwards, he believes that 80 percent of medical recommendations are crap technically speaking. Here he is, a veteran of the US army. He served in the US army. He is a board certified family physician. He’s the author of revolutionize your health with customized supplements and the founder of Revolution Health Dot Org. Dr Edwards, welcome to the show, man. It is good to be here. I’m excited for this topic today. You’re, you, you’re already, you’re already teasing it. You’ve been, you’ve been on a different level of excited since you’ve walked into, you know, this, this topic right here. Okay, so let me, let me kind of lay a little bit of foundation. Uh, this topic right here, I believe is probably one of the biggest lies propagated in medicine for the last 70 years. 60, at least 60 years.

Speaker 1: 01:56 He guess lies one of the biggest. Yes, I believe this is a, uh, a major load of crap. This is probably, you know, I would make this public enemy number one of the pure crap that we’ve been fed and said that this is a problem and here’s what we need to do about it. It, and it’s just gotten worse and worse and worse and worse, and there’s lots of evidence and there’s absolutely no way to sum all of this up in one podcast. It’s just not going to happen. But, uh, I did want to get a little bit of an intro to this topic because it’s something that I see so commonly. Um, so anyway. Well, let’s see. First, how many people is this effect? How much does this issue that this hot topic, who, who should really be paying attention right now? Who, who should listen to this?

Speaker 1: 02:44 Well, I’ll tell you, everybody with a pulse, everybody with a pulse. Okay. All right. And let me give you a little bit of background on this. So it’s gonna. It’s going to give out the answer of why this is a problem. So my wife and I, I’ll call, I’ll call it my new son now. So of my stepsons I’m 12 years old, went to his doctor for a routine exam and the doctor said, okay, well we’re going to check for no reason, and there’s. There was no medical problem. There was nothing going on. He just said, okay, well let’s go ahead and, and we’re going to do. We’re going to check her cholesterol levels. And My, my wife came home and told me that they were checking his cholesterol levels. Now you got to understand that, that this kid runs cross country. He’s 12, he runs cross country, he’s very fit. He’s very active. Has, I mean he’s a 12 year old kid is nutrition, didn’t the best but I’m not real focused on his nutrition but,

Speaker 1: 03:37 and he’s, he’s pretty fit and he’s healthy. There’s nothing wrong with him. And they said, we’re going to check her cholesterol levels. And I about flipped out. Why? Why would you check cholesterol levels in a 12 year old? Why? And the. She said, well, I, I don’t know. I said if, if he comes back with elevated cholesterol levels and they want to put him on a statin medication, then fire him. And I, I can’t even begin to tell you how passionately I feel about this and how unbelievably this is. This constitute medical rape as far as I’m concerned. Sure. It, it Asinine, absolutely asinine. Why would you check a level on someone that you’re not going to do anything about? And it means nothing. In a 12 year old, I’ve had pediatricians tell patient, young, young kids that, oh, your cholesterol is a little bit high. We should probably put you on a Staton. Are you freaking kidding me? What is wrong with you? So putting again, I can’t. I can’t even begin to go into all of this in one package.

Speaker 3: 04:46 No, this is great. This is. This is sheer passion in the sheer a misconception and a misunderstanding of what is contributing to optimal health. And it’s not just optimal health, but you know, cholesterol being this hot topic of a series of podcasts that I can imagine that we’re going to get into. So we’re talking about cholesterol and why is a. You talk about the pediatricians prescribing a stat and medications. What is the stat and medication for anybody that needs to be brought up? Suspense. Great Point.

Speaker 1: 05:19 So in order to answer that question, let me get into a little bit of biochemistry. I don’t want to get geeked out on it, but basically your body, in fact, one of the papers that came across my desk today talked about your cholesterol and your body is a product of two things. What you, what you make in your body, what we call endogenous production, and what you consume in your diet or what’s absorbed, and then kind of what’s reabsorbed. So your body is capable of making virtually all the cholesterol it needs. And we have a process through which that’s done, the rate limiting enzyme, meaning that the enzyme that controls the production of cholesterol the most, it’s the rate limiting step. If that enzyme is up, or if that enzyme is down, it’s gonna make the biggest difference in what is produced. And that enzyme is called HMG Coa reductase, Staton staton medication.

Speaker 1: 06:11 So these are like [inaudible] store and the, and the generic equivalents which are a tour of a Staton, Staton, staton. Uh, all of those kinds of drugs, those are hmg Coa reductase inhibitors, those medications block or reduce the activity of that enzyme and it’s important to understand the biochemical effects of doing that. And this is something that very few physicians in this country understand. My understanding is that in Europe they have a much better grasp of it and they manage their patients much more appropriately. And I’ll get into a little bit of that and we may have talked a little bit about some of this I don’t remember, uh, in, in previous podcasts, but the bottom line is, is these medications are medications that you take to block or reduce the activity of that enzyme, which ineffective lowers your cholesterol. The theory is that when we lower your cholesterol, it reduces your risk of cardiovascular disease.

Speaker 1: 07:11 Now I’ve got multiple problems with that statement because I think that is a yet another load of crap. And I think that’s probably been one of the biggest lies that’s been propagated in this country for years. It is not cholesterol. There are other problems and cholesterol is involved with the process. And often in some patients we see elevated levels of cholesterol or I’ll say altered lipid protein profiles, so your cholesterol levels and you’ve got a few of them, you got the quote, bad cholesterol, which is ldl or low density lipoproteins, and you have hdl, which is high density lipoproteins. So the. But the interesting thing about this is when we say your cholesterol is elevated, but your good cholesterol, your bad cholesterol, cholesterol as a sterol molecule, and it’s. It’s actually required for optimal health. There’s a condition known as Smith Lindley Outlet Syndrome, and if you are a homozygous abnormal meaning both gene copies from mom and from dad are abnormal.

Speaker 1: 08:13 It’s essentially incompatible with life. I’m not sure that I. I didn’t do the research for this part, particular part for the show, but I’m not sure that anyone has been born homozygote abnormal first Smith Lindley, orbitz, but you can be heterozygous abnormal meaning one copy is good, one copy’s not. Those patients are virtually incapable of producing cholesterol and it’s incompatible with life. You have to have cholesterol. You know, I think Dr Perlmutter says that 25 percent of your brain volume is cholesterol. Cholesterol is the backbone for vitamin D. Your vitamin D is directly derived from cholesterol is the backbone for all of your steroid hormones. It’s the for aldosterone which regulates sodium and water in your body for Cortisol, which is an essential hormone released from the adrenal glands in response to stress, and it does a number of things. It’s the backbone for Dah, Da Testosterone. How many times we’ve talked about testosterone, Estradiol, testosterone, all of those are derived from cholesterol.

Speaker 1: 09:14 Cholesterol is essential for the, what you call the fluidity of the cell membrane. Without that, you can’t, you can’t have normal cell function. It’s essential for bile salts. It’s a backbone for a bile, which is part of how we absorb fats and those kinds of things. There’s a guy named Duane graveline. He was an air force astronauts, uh, air force flight surgeon and an astronaut, and he went on a Staton and suffered what was called transient global amnesia where he lost his mind. He couldn’t, brain didn’t work, went off the Staton and specifically lipitor. It went off the Staton and got, I mean he, his brain function came back, went back on it again, transient Global Amnesia again and came off of it again and then ended up writing a book called lipitor thief of memory. So this is when you and when you listened to Dr Stephanie Seneff work on cholesterol, she thinks that that statens on.

Speaker 1: 10:09 I said on cholesterol. It’s on status. When you look at her work, she thinks that statins are so poisonous that no one under any circumstances should ever be on them. I don’t know that I’ll go quite that far, but I think she’s onto something there. When you look at the side effects that patients report and so often, and I’m not, I’m not faulting any other physicians in this. This is what I did when I was practicing traditional medicine and I put someone on a medication or they were on a medication and they would come in and they would say, I’ve got a problem with this or my muscles are achy specifically with this drug. What I did was drew a blood level CPK and I would look at that and look at their liver functions because this data is going to, you know, they, they work in the liver on this, uh, hmg Coa reductase pathway.

Speaker 1: 10:55 And then I would check the CPK and if that was normal that I would say, well, your muscles aren’t screwed up because of the statens your, you know, your labs are normal. Well, I’ll reference you to episode number two where we talk about normal versus optimal, what defines normal, these kinds of things. And then just because that lab is normal doesn’t mean you’re not having a problem. This is very, very simple. You come off the medicine, symptoms go away, you go back on medicine, your symptoms come back. It’s associated with the medicine. It’s a scientific, uh, it’s called Koch’s postulates, Koc, h, s, um, you can look that up. Maybe we should talk about that at some point. Um, but there’s a, there’s an association there, you, you’re on it, you have it, you’re not on it, you don’t have it. You can directly associate those two.

Speaker 1: 11:42 There’s a problem with that. Drug that’s not everyone, but I see it commonly. The second reason this, another reason that I’m that I rarely recommend drugs is because where they’ve proven to be beneficial is a much, much lower percentage of the population than what has been attributed, and we’ll talk more about this in the future, but so the cholesterol nonsense, I mean it’s just, it’s maddening and so it’s affected our diet. It’s affected, you know, you’re not supposed to eat eggs because there’s cholesterol in that. That’s bad cholesterol. It’s like this toxic molecule that’s going to cause you to blow up and yet we put glyphosate and freaking wheat and say, that’s okay. You know, eat all that you want, but something that’s essential for our health. We have. We got to give you a drug to stop. That is asinine. It’s absolutely asinine

Speaker 3: 12:34 for, for everybody that miss the podcast episode about glyphosate, the literally round up that they’re putting on your wheat. You got to go back a couple of episodes and listen to that, but it was a, you know, it’s the same thing is hey, this right here has been proven to be a poison only to yield more a wheat or you’ll yield more product to be able to sell for, for, for farms to sell. Right. Okay. But we’re very completely comfortable because it’s not a, at the forefront of our view in our understanding, but then you have something like cholesterol, which I would say by and large, most patients have no idea how cholesterol works. They just know that they’ve been told to stay away from it. Right. And a lot of, we’ll call it marketing companies for different big brand named food brands have come out with lower cholesterol products or foods or versions of it. And so yeah, absolutely. We’re getting into a, a Midi Harry

Speaker 1: 13:44 topic. Oh this. Yes. Absolutely. And the thing is, is it’s just, you know, I was looking up, you know, I love the concept of, and I say this all the time, you know, in 1984 or not in 1984 and the book 1984, George orwell talked about this, the concept of two plus two is five. And it’s illustrating the point of you hear a lie often enough, you begin to believe it. And at some point we’ll go into the history of the Diet heart hypothesis, how ancil keys started this whole thing in the 19 fifties, how initially he was laughed out of the room. He formulated what was called the seven countries study. It just, this, it was basically a lie, it was a misrepresent mess, representation of incomplete data. And he sold it as the truth and it was a political shift that allowed him to gain a foothold with a propagating this stuff forward.

Speaker 1: 14:40 And then you skip forward to the 19 seventies where, um, I was, I think it was mcgovern, senator mcgovern was in charge of the committee on food and all those things. And they came up with dietary recommendations and the crap that they came out with, those recommendations, not based on science that you know, the saturate 10 percent of your diet from saturated fat and you know, no more than 30 percent of fat in your diet and all that crap. It’s, it is not founded in science. And then, you know, we look at the data that’s coming out today is showing, oh well, you know, maybe, uh, maybe saturated fats not as bad as we as we think. And we think this is cutting edge science. Well, I’m going to go back to a study that was actually published this year in the British medical journal, but it references a study that was conducted from 1968 to 1973.

Speaker 1: 15:30 Interestingly, the data was never published and I don’t remember if we talked about this at all in a previous podcast or not. But uh, what they showed it was a randomized placebo controlled double blinded study. And you’re with like 9,000 participants. I mean, there’s a big study, and we’ll talk more specifically about this study, but um, I’m just going to quote this and their conclusions. It says, available evidence from randomized controlled trial shows that replacement of saturated fat in the Diet, which is what Ancil keys talked about in the 19 fifties. So if we replace saturated fat with linolenic acid, it effectively lowers serum cholesterol. So again, lowers cholesterol. That must be good. Well, who cares about. I mean, do you care about your cholesterol level? I don’t know what my cholesterol level is. Well, should you care? I don’t know. That’s what I’m trying to learn here today.

Speaker 1: 16:15 If, if we pulled the population, how many people do you think would say, yes, I care about my cholesterol? A large majority. And why? Why do you think we care about cholesterol levels? Do we care about cholesterol levels or do we care about something else? Uh, I think we only care about cholesterol levels as a society because, uh, because going back to that, you know, these big brand name food distributors have come out to market low cholesterol products, right? Because, and they came out with that because they saw it as a fad, and so it just perpetuated this, hey, cholesterol, uh, does x, Y, and Z to my body. Then, you know, I might as well choose a healthier option. Right? And how many doctors over the years have said, don’t eat eggs. It’s bad for you. Don’t eat saturated fat. Avoid that red meat that all causes heart disease.

Speaker 1: 17:07 You’re going to have a heart attack if you eat that stuff, you know, eat a time from what, 19, 84 or something like that. Had the, uh, the two eggs and the bacon underneath it. And we’re like, this is bad for you. Don’t eat this. And it’s just a load of crap. It’s not founded on science. And so it just gets over decades. We’ve been told this over and over and over again. And it’s a, it’s a pure load of crap. So I’m. So here they’re showing the saturated fat, replacing it with linoleic acid, lowers serum cholesterol. And I would argue that people couldn’t care less about their cholesterol level. They care about their risk for dying of a heart attack or a stroke. We’ve been, we’ve been, I would argue, we’ve been lied to that your cholesterol level predicts your risk of heart attack, and I would argue that that’s a load of crap going back to the 19 forties when they started the Framingham study.

Speaker 1: 17:56 The evidence is not solid and we’ll go into that in future podcasts. Let me finish this and then we’ll go to break. Um, so th, replacing with little lake acid rollover storm cholesterol, but does not support the hypothesis that this translates to a lower risk of death from coronary arteries or all causes. Findings from the Minnesota coronary experiment add to the growing evidence that incomplete publication has contributed to overstimulation of the benefits of replacing saturated with that with vegetable oils rich in linoleic acid. Again, it’s a load of crap. You’ve been lied to. You’ve been lied to. You’ve been lied to. I can’t say it enough.

Speaker 3: 18:36 We’ll take a quick break and when we come back we’ll learn a little bit more about what maybe is the underlying truth. Hear about cholesterol.

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Speaker 3: 19:17 Okay. We are back with Dr Chad Edwards and we’re talking about cholesterol, one of the hottest topics that we’ve covered, which is awesome, but a cholesterol, you know, it affects everybody that’s listening. Everybody with a pulse, as you said, and it’s such a crucial part of everyday. It’s required for human life. Absolutely. Okay. But maybe not everybody understands how it works. And so I want to just maybe take a step back and maybe you can walk us through why you know, the, the belief that cholesterol does affect the likelihood of a heart attack or a heart disease. It started somewhere. So maybe you walk us through what is commonly described as the relationship with cholesterol and the human body. What it does for the body.

Speaker 1: 20:09 Well, I can tell you that from my experience as a physician, what I heard for years and years and years was that high levels of cholesterol are associated with increased risk of cardiovascular disease. In fact, I wrote a little, I called it my smart book and it’s probably 200 pages that I am. It’s a very useful tool for me as a physician. And I basically, I took the data from something like the, uh, surviving sepsis campaign. It was an article, icu kind of stuff about really sick patients. And uh, there were a bunch of bullet points where there’s a bunch of stuff in this thing and you gotta follow these metrics. So I distilled that down into a one page list of bullet points and put it in this book and this is, this is how, you know if you sepsis follow these criteria, it’s it, it’s really kind of cookbook medicine, not that that’s bad and acute care medicine when there’s, I mean we have algorithms for how to deal with things and that’s not bad, especially in acute care medicine, in chronic medicine, different story, very, very different.

Speaker 1: 21:03 What I call biochemical individuality. Your physiology is very different than mine. The same set of rules do not apply to you. And what I see in my clinic is patients that have been to thousands of doctors, they’ve tried all these things. I still feel like crap. I still feel horrible. My doctor’s not listening to me. They tried this medicine, they won’t listen to this. They want and they come to me because they feel bad. And it’s because of biochemical individuality. And you have to understand biochemistry. You have to understand physiology, you have to understand pharmacology and pharmacodynamics and how it impacts each individual patient. And then you also have to get into, um, you know, ancestral type medicine and you have to get into nutritional medicine. How does what we eat impact our health, you know, all of these kinds of. And it’s very, it can be very complex and it can be overwhelming at times, you know, even for me.

Speaker 1: 21:47 And this is what I do everyday. Um, so when we look at this, this cholesterol component, I think it’s important to understand. So I’ve made some statements that were pretty strong that you’ve been lied to. That was probably the last one that we ended with. But so what gives me a foundation, I don’t want to go into a deep, but I just want you to understand that there is a basis for this. And so this is from another British medical journal thing. The conclusion from one study showed that high ldl cholesterol levels is inversely associated with mortality. And most people over 60, that means higher levels of Ldl have a lower risk of death, lower risk of mortality in people over 60. How many people have been told that by their physician? That’s a study, you know, a pretty, in fact that was a, um, a review of 19 cohort studies.

Speaker 1: 22:35 The data is all over the place in this and you’ve been told that it’s not that it’s cholesterol and again, we’ll get into, in a more detailed. I’m afraid I got out of two and mental tangent. Go. Let’s bring you back to where we’re supposed to go. No, no, no, no, no. This is good because this is really setting the stage for a few of the podcasts that are to come, but, uh, but go ahead and walk us through specifically the function and uh, maybe how cholesterol is expected to affect the likelihood of cardiovascular disease. Yeah. So, uh, they’ve basically what happened is when my understanding of the history is when, when they did studies looking at drugs that were not statens a lowering cholesterol. So these were things like Niacin, fibric, acid derivatives, different kinds of medications, and they gave them to patients with high cholesterol and they did in fact lower their cholesterol levels, but it didn’t change their death rate.

Speaker 1: 23:29 Um, there was the, I think it was the improve it trial that looked at Zevia, which absorbed which inhibits the absorption of cholesterol in the gut or slows it and by itself it didn’t make any difference and they thought, oh, this is going to lower cholesterol and people and it’s going to make it. Didn’t make any difference. Statins work by a different mechanism. They work by inhibiting HMG Coa reductase. Now, and I think we’ve talked about this before in another podcast, but three things are produced in that hmg Coa reductase a, a pathway. The first one is cholesterol. So we think that we give you a medication that lowers your cholesterol and, and it’s going to lower your risk of cardiovascular disease. We’ve already shown that that’s not true. There are multiple steady. Let Me Shit, let me rephrase that. There are multiple studies that would argue that independent of statens the data just doesn’t support it.

Speaker 1: 24:18 You lower cholesterol and you know, it doesn’t change things. And in fact, one study shows that patients presenting to the emergency department that are presenting with a heart attack, half of them, 50 percent of them have normal cholesterol levels. So. Well, of course the response then is, oh, well then we need to look for lower cholesterol levels going back to you. Well that lie wasn’t good enough. We got to get deeper in our lie. You’re not looking at the big picture. People get out of the lie, start focusing on what is it that’s causing a problem? So the issue with statens is that they inhibit cholesterol production, but they also inhibit cocuten. And I think that’s where a lot of the, uh, the side effects coming in. And like I said in Europe, they kind of have this figured out, anybody on a Staton, whether you take it appropriately or inappropriately, if you’re on a stat and you have to be on cocuten because your body is no longer capable of making appropriate levels of Cocuten, you have to have that stuff.

Speaker 1: 25:09 It’s very important antioxidant. And it’s also important for energy delivery. You gotta take cocuten for the love of all. That’s holy, take co q 10, but the last thing is that, that, uh, hmg Coa reductase pathway also produces a chemical called GGP, which activates a very, very inflammatory chemical called row. So when we inhibit HMG Coa reductase, you’re inhibiting cholesterol cocuten and inflammation through this chemical called row. Uh, Chris Masters, John has some amazing information, uh, done a lot of research into these pathways and that right there, the inhibition of this chemical called row explains a lot of the benefit of statens statens do reduce the risk of cardiovascular events, but the effect is not nearly as big as we think the effect has been proven on, on low levels, but it’s been proven in middle aged men that have already had a heart attack, what we call secondary prevention trials.

Speaker 1: 26:07 The trials that show consistent benefit are those, but the number needed to treat. In other words, how many people do we need to put on a stat and to, you know, to, to have benefit to produce 100, prevent one heart attack. Maybe the 12 year old, right? Uh, the, I’ll come back to that point in a second, but uh, because it’s an important point, um, the, uh, I mean, you, you have, depending on the study, you know, you’re talking 80 to 120 people to prevent one event, that these are high. It’s not like you put three people on a stat and then you’re going to prevent a heart attack. It’s not that we put you on a statin and we prevent a heart attack and you, the data doesn’t show it. They’re trying to. And then, you know, when we, when we see higher risk, oh, we got to drive that cholesterol down lower by giving them more staton.

Speaker 1: 26:52 I think cholesterol as a surrogate marker, I think it’s an innocent bystanders just sitting by, you know, uh, I didn’t do anything. Uh, it’s an innocent bystander or a scapegoat. I think the issue is this inflammation through which, uh, the statens inhibit. And so the more we inhibit that, the lower risk for cardiovascular disease. But I think the effect of that is independent of cholesterol. So anyway, so going back to that point about the 12 year old statens are contra indicated during pregnancy. You cannot be on those medications during pregnancy. Why is that the case? Because cholesterol is essential for life. You have to have it and it’s going to screw up. The fetus can’t. I mean this, this doesn’t make sense. This doesn’t. This isn’t optimal physiology that I answer the question where you were wanting to go with that.

Speaker 3: 27:43 Yeah, absolutely. So, um, what would you say about. I feel like a lot of patients are told, hey, stay away from the, from the cholesterol, it’s going to block your arteries.

Speaker 1: 27:56 Yeah. So the, there are multiple studies. In fact, there’s one that came across my desk today, why dietary cholesterol is no longer enemy number one. And there’s three points that they, um, that they, uh, that they made a, whether there’s actually a few. When you look at a high cholesterol diet, um, let’s see. First examine the effects of high cholesterol diet on the level of one of the main cardiovascular risk factors. LDL cholesterol will again, I’m not convinced that ldl causes heart disease. In fact, in patients over 60, it’s inversely proportional. So it goes back to what’s the foundation of this? Uh, there’s not a solid biochemical mechanism for, for, for this. Uh, and um, there’s just study after study that shows there’s just not a strong correlation with this. There are some in some studies, there may be some, some suggestion, but I don’t believe that the evidence is, um, is solid on one of the papers that I read today on the, on specifically on butter and that saturated fat piece.

Speaker 1: 29:00 Uh, they had three foundations in. The first one was butter is not necessary for maintaining good health, so they don’t consider it a health food. Second Butter is one of the foods with the highest saturated fat content and consuming on a regular basis promotes an increase in blood cholesterol levels. So if you presume that saturated fat increases cholesterol and cholesterol causes heart disease than you, their theory is that you can infer that saturated fat causes heart disease. But this, you know, this Minnesota coronary, a study that was published in the British Medical Journal suggest that’s not the truth, that while they lowered cholesterol, I think it was 16 percent, it actually, they actually had a higher risk of cardiovascular disease. So the studies on that independent of statens, the studies on the cholesterol stuff is just the, the data is all over the place. There is no consistency.

Speaker 1: 29:57 I can’t make a good recommendation to someone to lower their cholesterol and it’s going to prevent their cardiovascular disease independent of statens. So is there some, is there something else, and maybe there’s a followup podcast episode, but is there something else that maybe cholesterol is taking the fall for? Absolutely. And I think it’s, I think it’s really three things. Inflammation, oxidation and glycation. Glycation is blood sugar, you know, attaching to, it attaches to Ldl, uh, that, um, that, that compound, that chemical, that or that molecule that carries cholesterol, fats, those kinds of things. When that becomes glycated, it’s proinflammatory and oxidizes. So now you have a molecule that’s glycated that’s oxidized and inflamed. I think if you want to lower your risk of cardiovascular disease, one of the best things that you can do is optimize your blood sugar levels and insulin sensitivity. Do anything you can to reduce overall inflammation that goes into nutritional medicine.

Speaker 1: 31:03 If you are eating foods that are pro inflammatory, you know, we talked about glyphosate, we talked about gluten and I’m not trying to vilify them 100 percent for this, but if you are eating a food that causes some inflammation on an individual basis and it’s causing some inflammation and that may be any number of things. It could be dairy. For some people it could be a, they’ve got a food allergy to something and that’s causing inflammation. That inflammation is affecting all of this which increases your risk of cardiovascular disease. And if you’ve got, you know, uncontrolled allergies, independent of nutrition, anything that’s causing inflammation, it’s a shotgun effect. And so it affects a lot of things. And then oxidation. You know, when you look at marathon runners, they have a higher risk of cardiovascular disease than shorter duration. You know, if you run five k’s or 10 k’s every weekend, that doesn’t seem to have the same correlation.

Speaker 1: 31:55 Robb Wolf talked about that on a recent podcast about, um, you know, the, what they’ve seen. He was a study that he was quoting where when you get up into the marathon level, it’s because of oxidation. You are, it’s a very aerobic exercise. You’re breathing a lot of oxygen and every time you breathe oxygen, you create oxygen, free radicals, those free radicals or oxidative and you need good levels of antioxidants. So when you’re looking at trying to prevent cardiovascular disease, I would argue that, and I’m, I’m basing this not on a ton of randomized placebo controlled double blinded studies. I’m, I’m, I’m, I’m making the statement based on looking at the biochemistry, looking at all of the studies that are out there, trying to weed through all that and say the truth without big Pharma coming into my backyard and saying, well, this is how you. It’s almost like the devil sitting on your shoulder telling you what to think. And without that influence, it’s inflammation. Glycation and oxidation. You want to prevent cardiovascular disease, focus on those three things. So reduce inflammation, reduce oxidation, and reduced glycation inflammation is affected by status. And I, again, I, I, I do not think status are the solution for everything. I think they are far more problematic than we think, uh, but they are beneficial for some patients.

Speaker 3: 33:14 Boom. So as we get into it, we are talking about cholesterol. This is really kicking off a series of podcasts to come and it’s going to be exciting. I feel like I feel like maybe moving forward I need to, uh, just maybe plug a couple, you know, a couple claims that may get you fired up during the podcast because I don’t know if you’re excited enough about this one. Uh, you know, this is just one that gets me fired up. No, that’s great. And I know that all of the listeners, this affects everybody that’s listening here and so if they want to learn more about the different things that we’re talking about on the podcasts, where can they go? Well, I’ve got information on my website, revolution health.org.

Speaker 1: 33:57 Uh, there is a plethora of information on space doc.com. That’s space dock SBAC e doc.com. Uh, that’s dwayne grave lines, a website. Dr Stephen Sinatra is a cardiologist, has got a book called the Great Cholesterol Myth. Steven, I think it’s Kendrick has a book. Malcolm Kendrick has a book called the great cholesterol con. All of them referenced the studies that referenced the data, uh, and they are going against this mainstream. That cholesterol causes cardiovascular disease. Lots of information out there

Speaker 3: 34:29 for everybody listening, make sure that you check out revolution health.org. Go ahead and subscribe to the podcast on itunes and download, uh, the most recent podcasts@againstthegrainpodcast.com Dr Edwards. Thank you so much for joining us. As always. Thanks Marshall. Appreciate you. Thanks for listening to this week’s podcast with Dr Chad and tune in next week where we’ll be going against the grain.

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